ARID3A


ARID3A

AT rich interactive domain 3A (BRIGHT-like), also known as ARID3A, is a human gene.cite web | title = Entrez Gene: ARID3A AT rich interactive domain 3A (BRIGHT-like)| url = http://www.ncbi.nlm.nih.gov/sites/entrez?Db=gene&Cmd=ShowDetailView&TermToSearch=1820| accessdate = ]

PBB_Summary
section_title =
summary_text = This gene encodes a member of the ARID (AT-rich interaction domain) family of DNA binding proteins. It was found by homology to the Drosophila dead ringer gene, which is important for normal embryogenesis. Other ARID family members have roles in embryonic patterning, cell lineage gene regulation, cell cycle control, transcriptional regulation, and possibly in chromatin structure modification.cite web | title = Entrez Gene: ARID3A AT rich interactive domain 3A (BRIGHT-like)| url = http://www.ncbi.nlm.nih.gov/sites/entrez?Db=gene&Cmd=ShowDetailView&TermToSearch=1820| accessdate = ]

References

Further reading

PBB_Further_reading
citations =
*cite journal | author=Kortschak RD, Tucker PW, Saint R |title=ARID proteins come in from the desert. |journal=Trends Biochem. Sci. |volume=25 |issue= 6 |pages= 294–9 |year= 2000 |pmid= 10838570 |doi=
*cite journal | author=Kortschak RD, Reimann H, Zimmer M, "et al." |title=The human dead ringer/bright homolog, DRIL1: cDNA cloning, gene structure, and mapping to D19S886, a marker on 19p13.3 that is strictly linked to the Peutz-Jeghers syndrome. |journal=Genomics |volume=51 |issue= 2 |pages= 288–92 |year= 1998 |pmid= 9722953 |doi= 10.1006/geno.1998.5259
*cite journal | author=Suzuki M, Okuyama S, Okamoto S, "et al." |title=A novel E2F binding protein with Myc-type HLH motif stimulates E2F-dependent transcription by forming a heterodimer. |journal=Oncogene |volume=17 |issue= 7 |pages= 853–65 |year= 1998 |pmid= 9780002 |doi= 10.1038/sj.onc.1202163
*cite journal | author=Peeper DS, Shvarts A, Brummelkamp T, "et al." |title=A functional screen identifies hDRIL1 as an oncogene that rescues RAS-induced senescence. |journal=Nat. Cell Biol. |volume=4 |issue= 2 |pages= 148–53 |year= 2002 |pmid= 11812999 |doi= 10.1038/ncb742
*cite journal | author=Kaiwen M |title= [Involvement of E2FBP1, an ARID family member protein, in the p53 regulatory pathway] |journal=Kokubyo Gakkai Zasshi |volume=69 |issue= 2 |pages= 152–61 |year= 2002 |pmid= 12136662 |doi=
*cite journal | author=Goebel P, Montalbano A, Ayers N, "et al." |title=High frequency of matrix attachment regions and cut-like protein x/CCAAT-displacement protein and B cell regulator of IgH transcription binding sites flanking Ig V region genes. |journal=J. Immunol. |volume=169 |issue= 5 |pages= 2477–87 |year= 2002 |pmid= 12193717 |doi=
*cite journal | author=Strausberg RL, Feingold EA, Grouse LH, "et al." |title=Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences. |journal=Proc. Natl. Acad. Sci. U.S.A. |volume=99 |issue= 26 |pages= 16899–903 |year= 2003 |pmid= 12477932 |doi= 10.1073/pnas.242603899
*cite journal | author=Ma K, Araki K, Ichwan SJ, "et al." |title=E2FBP1/DRIL1, an AT-rich interaction domain-family transcription factor, is regulated by p53. |journal=Mol. Cancer Res. |volume=1 |issue= 6 |pages= 438–44 |year= 2004 |pmid= 12692263 |doi=
*cite journal | author=Fukuyo Y, Mogi K, Tsunematsu Y, Nakajima T |title=E2FBP1/hDril1 modulates cell growth through downregulation of promyelocytic leukemia bodies. |journal=Cell Death Differ. |volume=11 |issue= 7 |pages= 747–59 |year= 2005 |pmid= 15017387 |doi= 10.1038/sj.cdd.4401412
*cite journal | author=Grimwood J, Gordon LA, Olsen A, "et al." |title=The DNA sequence and biology of human chromosome 19. |journal=Nature |volume=428 |issue= 6982 |pages= 529–35 |year= 2004 |pmid= 15057824 |doi= 10.1038/nature02399
*cite journal | author=Nixon JC, Rajaiya JB, Ayers N, "et al." |title=The transcription factor, Bright, is not expressed in all human B lymphocyte subpopulations. |journal=Cell. Immunol. |volume=228 |issue= 1 |pages= 42–53 |year= 2004 |pmid= 15203319 |doi= 10.1016/j.cellimm.2004.03.004
*cite journal | author=Rajaiya J, Nixon JC, Ayers N, "et al." |title=Induction of immunoglobulin heavy-chain transcription through the transcription factor Bright requires TFII-I. |journal=Mol. Cell. Biol. |volume=26 |issue= 12 |pages= 4758–68 |year= 2006 |pmid= 16738337 |doi= 10.1128/MCB.02009-05
*cite journal | author=Olsen JV, Blagoev B, Gnad F, "et al." |title=Global, in vivo, and site-specific phosphorylation dynamics in signaling networks. |journal=Cell |volume=127 |issue= 3 |pages= 635–48 |year= 2006 |pmid= 17081983 |doi= 10.1016/j.cell.2006.09.026
*cite journal | author=Lin D, Ippolito GC, Zong RT, "et al." |title=Bright/ARID3A contributes to chromatin accessibility of the immunoglobulin heavy chain enhancer. |journal=Mol. Cancer |volume=6 |issue= |pages= 23 |year= 2007 |pmid= 17386101 |doi= 10.1186/1476-4598-6-23

External links

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  • ARID3A — HUGO 3031 Símbolo ARID3A Símbolos alt. BRIGHT, DRIL1, DRIL3, E2FBP1 …   Wikipedia Español


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